Monday, March 29, 2010

High, Soft Cervix, But No Egg White Mucus

hannibal_tajima @ 2010-03-29T18:35:00

Depression

definition
 
mental illness is one of mood disorders as affective disorders
 = Frequent fluctuations of mood and drive there
 Stay feelings of sadness and loneliness for a long time, can state to develop depression
 For example, after death of a close relative
 often associated with feeling of deep sadness
 Many tell of agonizing emotional emptiness
 feel throughout their life strongly affected
 succeed often difficult to perform daily tasks
 strong self-doubt of interest + no longer for things that had them previously important
 distraction + encouragement give the other no relief
frequency
 One of the most common mental disorders
 risk is 16 to 20 percent
 affects women more often
can occur at any age it
  The most common between the ages of 18 and 25
 After a depressive phase is about half of those affected developed depression again

causes
 Caused by the interaction of different biological and psychological causes
 if increased susceptibility is able to stressful life events have a depressive illness cause
 Example:
• long-lasting excessive demands in the workplace
• relationship problems
 In these cases we speak of a primary depression
 secondary depression is triggered by other mental or physical illness
Biological factors

 The trigger or risk factor of primary depression -> Features in Setup + activity of the brain into question

  example in depressed people are in the brain regions responsible for development of objectives and the emergence of positive emotional states are less active control
 brain areas that behavior in new situations + by social contacts -> much less pronounced
 brain areas involved in the generation of negative emotions -> over active
 brain structures that regulate the release of stress hormones -> overactive
 Some special features in brain metabolism:
 serotonin and norepinephrine available to a lesser extent
 play in the development + regulation of emotions important role
 Notes genetic predispositions:
increased  risk if there are close relatives already diagnosed with depression or were
 physical ailments depressive disorders cause
 Example:
• epilepsy or thyroid disease
 connection with drugs that are used as a cancer treatment be -> A triggering depression

Psychological factors

 negative thought patterns that were acquired in childhood -> often responsible for primary depression
 negative experiences with previous caregivers
 disregard by parents or violence
 often lead to feelings of self-rejection + development negative self-image
 affected tend to their environment to deal with suspicious and pessimistic
 In consequence, they suggest even neutral events as negative + as confirmation of their worldview
 New positive events - have not> as such perceived
 vicious circle that is maintained by looking at the world

 Another factor is dealing with failures
 before their depression often experienced situations where it was not possible to influence circumstances or control
 The result was Feeling of helplessness
 feeling is reflected -> very passive in dealing with problems
 behavior do not see themselves in a position to influence their lives positively

 tendency for increased vulnerability to negative events to this cause alone with him to seek
more explanations

 try to explain how depressions arise + as they are maintained
 set scale of positive experiences in the center that someone play experiences in response to behavior
 two factors central role influencing
 circumstances of people , how often positive emotions such as joy or satisfaction experienced
 own behavior influences degree of pleasant consequences:
• Someone who can go to other forms, obtained, for example, more positive feedback from other people
• Depressed behavior -> withdrawal and passivity -> ; negative feedback + failures
• boosts the depressive behavior

symptoms
 typical symptoms: depressed mood
 
loss of interest
 sadness, sadness, helplessness, hopelessness
 
fears, guilt
 impaired concentration
 withdrawal, avoidance
 reduction of the drive
 In severe depressive symptoms:
 crippling feeling of numbness
 The mood is largely independent of external influences
 successes or enjoyable activities - work> no mood improvement
 often listless
 most pessimistic towards the future with confidence
 
Often thought of suicide often withdraw from social environment
 disturbed sleep and appetite
 feel in physical expression (movement, facial expressions) inhibited
 Some find it difficult to sleep + have to move constantly
can  strong anxiety + irritability thereby be
 themselves blame them on relatively minor incidents + develop an irrational fear of possible consequences
 Some are affected by many physical complaints
 dizziness, headache, throat Kloßgefühl
 no medical explanation for
 In rare cases, severe psychotic symptoms:
 hear such voices they abuse or developing delusions

diagnosis
 diagnosis is made when:
 present two of the core symptoms
• depressed mood
• loss of interest
• joylessness
• Drive reduction
 the person in two other symptoms suffered
• sleep
• fears
• impaired concentration
 These symptoms occur at least two weeks
 Depending on how many symptoms are present:
 light, a moderate or severe depression
 single phase of depressive symptoms -> depressive episode
 repeated episodes -> recurrent depressive disorder
 Some symptoms are not strong enough to be able to detect a depressive episode -> the depressive state but holds on for years on
 is it is a dysthymia
 The doctor or psychologist + in-depth discussion
 shows how many symptoms of depression are present
serve  So-called interview guidelines to assist
 series of questionnaires that capture whether and to what extent depressive symptoms in the past have existed
 including physical and neurological examinations
 diseases such as Alzheimer's and dementia and cardiovascular disorders and infectious diseases as a cause

therapy
 excluded as biological and psychological causes -> therapy usually two levels:
 medical
 psychotherapy
 combination increases the probability of success Medical treatment


medical therapy   focus
antidepressants, which act the balance of brain chemicals influence
 in regulation only after two to three weeks
 lead in start time increased to severe side effects
 Fatigue
 nausea
 first detected after the time that drug works
 If not, or can be side effects after the first two to three weeks after
 treatment with another antidepressant useful
 After resolution of symptoms antidepressants should have a time be taken further to avoid long
relapses
  If drugs are sold -> Symptoms may occur again
 In addition to drug therapy, yet: sleep deprivation
 
Light Therapy •
is mostly used for winter depression occurring (Known as seasonal depression)
 SSRI and SNRI
 In the case of drug treatment of depression with and without anxiety symptoms are often used:
• selective serotonin reuptake inhibitor (SSRI)
• Citalopram
• fluoxetine
• Serotonin-norepinephrine reuptake inhibitors (SNRI) used
• Venlafaxine
 SNRIs and SSRIs specifically inhibit the resumption of brain chemicals serotonin or serotonin and noradrenaline in the nerve cell
 This extension of positive action
 lightens the mood + alleviates fears
 Rare side effects:
• Cardiovascular complaints
• Headache
• nausea
• digestive problems
 SNRI should not be taken with triptan drugs
 Are SNRIs such as venlafaxine after resolution of depressive symptoms have taken longer -> rare relapses
 Other antidepressants
 MAO inhibitors such as moclobemide and tri-and tetracyclic antidepressants such as doxepin
 prevent the degradation of certain brain chemicals
 This are chemical messengers in higher concentrations -> alleviate feelings of anxiety and depression
 tri-and tetracyclic antidepressants resolve anxiety and reassure
 affect the concentration of neurotransmitters in the brain -> Record in the nerve cells resistant
 messengers in higher concentrations for transmission between nerve cells available
 benzodiazepines
 bridge to antidepressant effect -> in some cases so-called benzodiazepines -> ; treatment of depressive symptoms
 soothe and promote sleep

Psychotherapeutic treatment
 goal -> depressive behaviors and thought patterns to reduce and replace them with positive
 against activity and loss of interest and the associated Lack of positive experiences to address
 develop a more active day structure
 aware enjoyable activities into daily schedule schedule
 regular and active daily routine -> Lower withdrawal behavior
 again a positive experience + discover lost interest again with increasing time
 initial hard make life more active again
 In time, however, most felt that the activation brightens their mood for
 social environment -> communication exercises and role-plays - 'positive interactions with other people (social skills training)
 The negative thought patterns Another approach
 express themselves in a negative world view, self-depreciation or self-doubt
 Psychotherapy works out whether they are consistent with the reality
 reviewed how this will affect the own-being
 then develop common realistic ways of thinking, the negative thought patterns to counter particularly
 severe depression -> any therapy outpatient hospital
 Light and moderate depression in general.
 After the acute symptoms of depression -> make sense in a deep psychological Treatment causes get to the bottom

course
 course different
 depends on various factors
 decisive factors are the number of previously experienced depressive episodes, the length of these episodes and the severity of depression
 depressive episode lasts usually about five months reach
 time between periods of depression on average about four to five years
 Up to two thirds of those affected substantial improvement to the healing of chronic
 -> 10 to 20 percent
 15 percent -> suicide
 prognosis of sex of the person concerned + Influenced the course:
 women have an increased risk of suffering from depression
 a young age already sick or have already experienced a depressive episode -> at risk of experiencing a further episode of depression
 especially important that those affected by drug + maintenance therapy psychotherapeutic strategies prevent
 75 percent of the cases - support> other symptoms, a depressive disorder:
 physical illnesses such as asthma and cardiovascular disease
 mental disorders such as anxiety disorders, eating disorders and substance abuse prevention


help  Certain practices prevent depression
 praise for small successes and value
 activate themselves and positive plan activities
 positive thoughts
 particularly important in difficult phases
 nobody
 protected distinguish often difficult to know if being normal reaction on critical circumstances, or whether they hold a depression
 symptoms despite improvements to -> professional help
 The later the treatment, the worse the prognosis
 solidify with time, negative thought patterns + Experience positive events and more rare.
 therapy carry out at least until stabilized and relapse risk was minimized apply
 conduct further + monitor own being continuously
can help  diary countermeasures
 first warning signs of a recurring depression quickly

© http://www. onmeda.de / diseases / depression.html ; 23.56 clock, Saturday, 10/04/2010, as of 25 June 2009